Articles written in Journal of Genetics
Volume 25 Issue 2 February 1932 pp 257-259
The mutation “white eye” is reported to have occurred once again in
A sex-linked recessive lethal, designated
Volume 26 Issue 3 December 1932 pp 351-358
An autosomal recessive eye colour purple in
It is suggested that the modes of interaction of these two genes, vermilion and purple, may be explained on the assumption that they are similar in nature and in function, and hence behave as allelomorphs.
Volume 29 Issue 2 July 1934 pp 269-276
Three purple allelomorphs of
Volume 30 Issue 1 January 1935 pp 15-29
1. Snapt, tilt and sepia are three genes on the left arm of the
2. Stubble, a dominant, and glass form the nucleus of the 3rd linkage group, and short4 and jaunty that of the 4th. Tangled, it is thought, belongs to the 5th.
3. It is suggested that: (i) a portion of the left arm of the 3rd chromosome of
4. It is suggested that the apparently disproportionate effect of some genes affecting eye colour is in reality cumulative.
Volume 30 Issue 2 March 1935 pp 233-241
1. Seventeen autosomal colour mosaics in the budgerigar (
2. The chromosome number is 50–60. There are three size classes: the
Volume 32 Issue 1 February 1936 pp 5-15
The character Plexus is described and shown to be based not on a single gene but on the inverted order of some of the genes in the
A series of mosaics arising in the Plexus stock is described and the origin and nature of the condition discussed.
Volume 34 Issue 1 February 1937 pp 91-96
Volume 37 Issue 1 December 1938 pp 211-228
Both types are found among the offspring of homozygous and of heterozygous Plexus females.
The haploid areas, in all but one case, carried the paternal genome.
The diploid areas of haplo-diploid mosaics having nearly always been female, must usually have been derived from the same male pro-nucleus as the haploid area. This indicates that in these cases the male pronucleus, in the first division after fertilization, has not yet united effectively with the female pro-nucleus. The latter either fails to divide at this division, or one of its daughter nuclei fails to become incorporated in a zygote nucleus and is eventually lost.
Double fertilization is probably occasioned by failure of the two daughter cells resulting from the second maturation division to separate from each other.
Volume 39 Issue 2 January 1940 pp 273-283
1. The case is reported of a chromosome termed
2. If the breakage and reunion occurred by a process of crossing-over, the chromocentral regions of
3. In its new position on the
4. When the
Volume 48 Issue 2 August 1947 pp 223-236
Spontaneous mutation frequencies were studied in a series of four experiments involving about 16,500 sperm obtained from seventy-nine male
The data show that both the overall mutation frequency and the distribution of lethals in terms of the flies’ age are affected by the rate at which the flies are allowed to breed. In intensive breeding, mutations were found only among the sperm of the first 24 hr. In slow breeding a well-defined periodicity was apparent: a first priod of high frequency was invariably followed by a period of low frequency which was in turn succeeded by a rise usually maintained till death.
The data suggest (1) that the first batch of mature sperm to be used is the most susceptible to mutagenic influences, and (2) that the effect of ‘storing’ the sperm under conditions of slow-breeding becomes apparent in the third week of life.
It is probable that the capacity of the individual male to produce sperm is not reduced by intensive breeding, but that intensive breeding may exhaust the supply in about one-third of the life span.
Volume 49 Issue 2 October 1948 pp 120-125
The sterility of certain
Volume 49 Issue 3 December 1949 pp 192-208
Analysis of the differences in fertility between an old marked stock (
The hypothesis that the cause of such divergence might be related to an increased mutation-rate connected with major mutations in general was tested by mutation-rate experiments in both stocks. The mutation rate of
The data suggest that mutations tend to occur earlier (i.e. either during embryonic development or during early germ-cell formation) in marked stocks than in wild stocks.
Observations in other fields are shown to be in line with the hypothesis, and its applicability to evolutionary phenomena is stressed.
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