Articles written in Journal of Biosciences
Volume 3 Issue 3 September 1981 pp 311-320
The interactions of flavin mononucleotide (riboflavin-5'-monophosphate) with two polypeptides, poly-(α-L-lysine) and poly-(α-L-histidine) in water and 0.05 M phosphate buffer were studied by measuring circular dichroism in the pH range 3 to 11. The interation of flavin mononucleotide with the two polypeptides was due to hydrophobic as well as ionic associations and was further influenced by the involvement of the ribityl side chain. The results of the present study have shown that small changes in the environmental conditions of the interacting molecules could modify their mode of interaction considerably.
Volume 12 Issue 1 March 1987 pp 55-59
The levels of lipoprotein-X in circulation increased with chronic administration of ethanol or acetaldehyde. A similar profile was seen in rat serum with alkaline phosphatase activity and bilirubin content. Total cholesterol, phospholipids and triglyceride contents increased followed by a decrease by progressive feeding with ethanol or acetaldehyde. The effect of acetaldehyde was more pronounced than that of ethanol.
Volume 13 Issue 3 September 1988 pp 269-274
The levels of lipid peroxides in circulatory lipoproteins increased with chronic administration of ethanol or acetaldehyde. Low density lipoprotein showed a greater increase in its content of lipid peroxides than very low density lipoprotein or high density lipoprotein. However, very low density lipoprotein was more prone to lipid peroxidation
Volume 14 Issue 1 March 1989 pp 55-61
Chronic administration of ethanol in rats caused the reduction of serum cholesterol binding reserve. The very low density and high density lipoproteins, main serum cholesterol binding reserves, were slightly increased with corresponding increases in their lipid and protein components during initial stage of alcohol consumption. However, these capacities get deminished during reversal of hyperlipemia induced by prolonged action of ethanol. This situation may be an early indicator for the initiation of hepatic damage and a variety of secondary effects of ethanol.
Volume 44 | Issue 5
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