Volume 30, Issue 4
October 1949, pages 185-242
pp 185-194 October 1949
pp 195-206 October 1949
pp 207-214 October 1949
pp 215-225 October 1949
One kind of asphyxial increase in excitability is inhibited by glucose and increased by iodoacetic acid and acid solutions.
The second kind of asphyxial increase in excitability is increased by glucose and inhibited by iodoacetic acid and acid solutions.
The mechanism, which produces the glycolytic asphyxial increase in excitability is antagonistic to the non-glycolytic one.
Glucose is utilised anaerobically in two ways; one of these is the same as that in which it is utilised ærobically, and the other is antagonistic.
Tone producing substances depress the response of asphyxiated muscle to alternating current if the latter is stimulated about once in 10–15 minutes.
There are two anaerobic mechanisms in acid solutions; one is antagonistic to and the other same as aerobic one,
During asphyxial increase in excitability, inhibition may be turned into contraction.
pp 226-242 October 1949
From the very early stages of development the orientation of the embryonic knob is antimesometrial.
The site of Implantation is clearly defined antimesometrially even before the embryo is actually attached.
The loss of the uterine epithelium cannot be attributed to the phagocytic activity of the trophoblast, though the trophoblast may ingest the degenerated epithelial ceil dêbris. Only later the trophoblast is seen to actually erode the uterine endometrium for estblishing placental relationships.
Amniogenesis occurs by cavitation. No distinction into primitive and definitive amnion can be made out, because they are one and the same; the delaitive amniotic cavity is differentiated when the amniotic cavity is formed.
The uterine glands do not undergo resorption, but are mechanically pushed towards the mesometrial side due to the widening of the antimesometrial part of the uterus.